Smart man. Yeah, those all day chemists sure are great. Oi, think I took a bit too much for my mescaline hcl microdose, feel a bit out of it, grant me some lenience.
The baclofen/liofen looks pretty affordable, only problem is that they don't offer faster shipping, and it can take pretty long to get there, then there's always the customs risk (probably low due to their shipping style drawing much less attention). But, once you factor in the conversion ratio to keep withdrawals away at the dose you (were) using, that you'll need to use it multiple times per day, probably many more than the usual 3x, plus the flat shipping price, I could see it getting pretty expensive.
It would probably be a good idea to buy it anyway to supplement what they'll (hopefully) give you. I really doubt they'll give you the dosage you'll need, I don't know if they'll even legally be able to, because it's so insane. They tend to be skimpy anyway.
Also, I think being labeled with "drug seeking behavior" may stay on your record and follow you, which will cause problems. Pretty unfortunate...
Another option: etizolam. Have tried benzos before? It's not a gaba-b agonist of course, and a thienodiazepine, but ithe latter isn't really relevant for this. They're also not cross tolerant, but drugs can have vastly different effects at different dosages, so who knows what phenibut's doing in you, and it would be smart to use a combo, adjusting dosages based on effects. Oh snap, I remember that allegedly gaba-b agonists are supposed to upregulate gaba-a receptors, and vice versa. Once again, at your dose, who knows what effect or how strong of an effect it's had. Volumetric dosing and start very low if you try this. IIRC etizolam has been shown to cause significantly less dependence and tolerance than other benzos, and it's also more selective for the anxiolytic A3 subreceptor, along with being legal, cheap, and feeling great, all making it the best choice IMO.
As for Oxcarbazepine, the mood stabilizer/anticonvulsant:
GABA-B
They can stimulate the opening of K+ channels which brings the neuron closer to the equilibrium potential of K+, hyperpolarising the neuron. This prevents sodium channels from opening, action potentials from firing, and VDCCs from opening, and so stops neurotransmitter release. Thus GABAB receptors are considered inhibitory receptors.
Oxcarbazepine
sodium channel inhibition (presumed to be the main mechanism of action) – and is generally used to treat the same conditions.
Anticonvulsants:
Conventional antiepileptic drugs may block sodium channels or enhance γ-aminobutyric acid (GABA) function. Several antiepileptic drugs have multiple or uncertain mechanisms of action.[2] Next to the voltage-gated sodium channels and components of the GABA system, their targets include GABAA receptors, the GAT-1 GABA transporter, and GABA transaminase
May end up being the best/healthiest/cheapest choice. Not sure if Oxcarbazepine, but someone used it specifically for phenibut withdrawal and said it worked.
I did way too much searching and reading on this yesterday, and I'm not very familiar with drugs or conditions relevant to me. Take over, hopefully I didn't forget something important.