I know what progestins are and I'm very familiar with how they work. That does not cite what you just said. Progesterone is made in genetic men via the adrenals and testes, where some of it is released into the bloodstream before being converted into androgens.

What I don't fully well understand is the adrenal-hypothalamic (I believe it's hypothalamic) axis. Hormones ARE regulated at the adrenal level and can be suppressed or stimulated etc.. I do know that all progestins suppress cortisol, which how can that be? Theres a few ways as I understand it: Medroxyprogesterone, cyproterone, desogestrel, drospirenone, etc all follow the same pathways endogenous progesterone does and are metabolized into their own derivates of cortisol (with the acetate still intact in cypro and medroxys case, i'm fairly sure), which are chemically defunct but are able to bind to GR/MRs and therefore reduce otherwise endogenous cortisol activity, OR...which makes more sense, progesterone production in the adrenals is suppressed, because I don't realistically see progestins' cortisol derivatives achieving that kind of coverage enough to interfere with endogenous cortisol.

The adrenals do follow a feedback, but it just isn't all too well clear to me. I mean it when I say I would love a source because I'd like to understamd this all a bit more.