Where should I go from here? Info in captions

Tons of guys in my Facebook group with 'high e2' (by your definition) and zero issues with prolactin, as well as zere issues with gyno.

What is 'my definition' of high E2?

This is our first ever interaction, so I'm not sure how you're sure of what my views are.

I have no idea why this is difficult to understand. I'm really quite blown away how such a simple concept is being immediately dismissed when I can demonstrate it until the cows come home.

I'm not dismissing anything other than your dismissal of the well defined fact that estradiol, being an ER agonist, possess the potential to contribute towards the development of gynecomastia. This is a fact.

Gynecomastia: Etiology, Diagnosis, and Treatment

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