Ah, sorry! I dumbed it down a little bit thinking you were a laymen. I didn't even wanna get into the fact duloxetine is an SNRI because most people don't understand. My post is still accurate in terms of the BDNF, as that is the true mechanism of action of all antidepressants as far as I see it. Correcting the deficiency in synapses through BDNF is more of a fix for depression, rather then just a band-aid. The thought behind SNRIs is that NE is also deficient in the synaptic cleft and through blockade of reuptake you get more activity. This blockade is but a fraction of what adderall can do. Adderall does do a tiny bit with serotonin but it mostly works through dopamine which cascades down to releasing NE which it also happens to block the reuptake of. This puts a lot of Dopamine and NE in the synaptic cleft which gets you higher activity and higher reward. As I'm sure you know, Dopamine is heavily involved with the reward pathways... Anyways! Adderall is only a band-aid for depression, and it hurts a lot when you take it off because your brain becomes dependent on it. BUT, people that do not lack the number of synapses will come out of it quickly once NT levels return to normal. I really think of it as your brain is just worn out after you've been on it for a while and needs time to make NTs. Your current therapy isn't really taking anything away from the antidepressant effects, but you are at great risk to go through swings of severe depression if you abruptly stop Adderall. If you just started, not much risk at all, but I'd consider cutting back and stopping it.
Do you get what I'm saying here though? Your brain is being stressed quite heavily for NE while you're on both of those meds, and when the Adderall dissipates, your already low baseline levels of NE are even lower. You really do not want to be on Adderall with depression. I know from learning about it in school and through rotations and my own interests. We've got a great PSYC guy that is very active at his practice. As for publications, I'm sure they're out there. If anything, your adjunct therapy should of been buproprion as per guidelines and I agree with that.
It does make sense your anxiety lifted a bit with an SNRI, they do show really good evidence (especially duloxetine), but I am not sure of their mechanism. That is an area that isn't nearly as understood, at least by me.
I hope this all makes sense, I would be happy to elaborate on anything if you'd like. I'm really knowledgable about depression due to class and I designed a drug that could in theory work as a fast acting antidepressant, which was pretty sweet (You can pubmed search fast acting antidepressant and you'll get a number of studies using a ketamine like drug).